lv scar heart | myocardial scar and lvef lv scar heart This study was conducted to assess the relationship between scar burden (extent and severity) and the follow-up left ventricular ejection fraction (LVEF). Methods. Patients were .
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0 · ring like left ventricular scar
1 · regional left ventricular scars
2 · nonischemic left ventricular scar
3 · myocardial scar and lvef
4 · lvef myocardial scar risk
5 · lge cmr left ventricular scar
6 · left ventricular scar mri
7 · left ventricular scar diagnosis
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ring like left ventricular scar
Myocardial scar and LVEF are both risk markers for all-cause and cardiac death in patients with nonischemic cardiomyopathy. However, whereas myocardial scar has strong and incremental prognostic value for SCD risk .Ventricular scars are a major cause of ventricular tachycardia in many forms of heart disease. Methods to identify and characterize scar hold promise for identifying patients at risk. Despite .
The identification of a nonischemic left ventricular (LV) scar represents a significant challenge for cardiologists due to the need for differential diagnosis among a wide range of diseases, with .
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Left ventricular (LV) scar on late gadolinium enhancement (LGE) cardiac magnetic resonance has been correlated with life-threatening arrhythmic events in patients with apparently idiopathic ventricular arrhythmias (VAs). We . The extent of left ventricular scar on LGE-CMR was characterized in terms of percent scar, scar surface area, and number of transmural scar segments. The primary end point was appropriate ICD therapy (as a surrogate . This study was conducted to assess the relationship between scar burden (extent and severity) and the follow-up left ventricular ejection fraction (LVEF). Methods. Patients were . The amount of replacement fibrosis in the LV myocardium (scar burden) measured by cardiac magnetic resonance (CMR) using late gadolinium enhancement imaging might be a key factor to individualize risk stratification .
Necrosis regions found in the heart (including left atrium (LA) pre-ablation fibrosis, LA post-ablation scar and left ventricle (LV) infarction), depending on the location and size, .Disease-causing genetic defects in arrhythmogenic cardiomyopathy according to predominant ventricular involvement. Mutations in DSP, PLN and FLNC genes are the most common gene defects causing arrhythmogenic left ventricular .
Scar quantification by CMR appears to independently predict clinical events and LV functional improvement, in both ischaemic and non-ischaemic cardiomyopathy, even when . Myocardial scar and LVEF are both risk markers for all-cause and cardiac death in patients with nonischemic cardiomyopathy. However, whereas myocardial scar has strong and incremental prognostic value for SCD risk stratification, LVEF has no incremental prognostic value over clinical measures.
Ventricular scars are a major cause of ventricular tachycardia in many forms of heart disease. Methods to identify and characterize scar hold promise for identifying patients at risk. Despite implantable defibrillators, which provide protection from sudden death, recurrent VT predicts increased risk of mortality and negatively affects quality .The identification of a nonischemic left ventricular (LV) scar represents a significant challenge for cardiologists due to the need for differential diagnosis among a wide range of diseases, with major diagnostic, therapeutic, and prognostic implications for patients and their families. Left ventricular (LV) scar on late gadolinium enhancement (LGE) cardiac magnetic resonance has been correlated with life-threatening arrhythmic events in patients with apparently idiopathic ventricular arrhythmias (VAs). We investigated the prognostic significance of a specific LV-LGE phenotype characterized by a ringlike pattern of fibrosis. The extent of left ventricular scar on LGE-CMR was characterized in terms of percent scar, scar surface area, and number of transmural scar segments. The primary end point was appropriate ICD therapy (as a surrogate for sudden cardiac death).
This study was conducted to assess the relationship between scar burden (extent and severity) and the follow-up left ventricular ejection fraction (LVEF). Methods. Patients were referred for viability assessment with late gadolinium enhancement (LGE) on cardiovascular magnetic resonance imaging. The amount of replacement fibrosis in the LV myocardium (scar burden) measured by cardiac magnetic resonance (CMR) using late gadolinium enhancement imaging might be a key factor to individualize risk stratification and refine patient selection for mitral valve interventions (12–14).
Necrosis regions found in the heart (including left atrium (LA) pre-ablation fibrosis, LA post-ablation scar and left ventricle (LV) infarction), depending on the location and size, can have various implications on the cardiac conditions of the patients.
Disease-causing genetic defects in arrhythmogenic cardiomyopathy according to predominant ventricular involvement. Mutations in DSP, PLN and FLNC genes are the most common gene defects causing arrhythmogenic left ventricular cardiomyopathy. LV, left .
Scar quantification by CMR appears to independently predict clinical events and LV functional improvement, in both ischaemic and non-ischaemic cardiomyopathy, even when accounting for clinical, electrocardiographic and LV lead characteristics. Myocardial scar and LVEF are both risk markers for all-cause and cardiac death in patients with nonischemic cardiomyopathy. However, whereas myocardial scar has strong and incremental prognostic value for SCD risk stratification, LVEF has no incremental prognostic value over clinical measures.Ventricular scars are a major cause of ventricular tachycardia in many forms of heart disease. Methods to identify and characterize scar hold promise for identifying patients at risk. Despite implantable defibrillators, which provide protection from sudden death, recurrent VT predicts increased risk of mortality and negatively affects quality .The identification of a nonischemic left ventricular (LV) scar represents a significant challenge for cardiologists due to the need for differential diagnosis among a wide range of diseases, with major diagnostic, therapeutic, and prognostic implications for patients and their families.
Left ventricular (LV) scar on late gadolinium enhancement (LGE) cardiac magnetic resonance has been correlated with life-threatening arrhythmic events in patients with apparently idiopathic ventricular arrhythmias (VAs). We investigated the prognostic significance of a specific LV-LGE phenotype characterized by a ringlike pattern of fibrosis. The extent of left ventricular scar on LGE-CMR was characterized in terms of percent scar, scar surface area, and number of transmural scar segments. The primary end point was appropriate ICD therapy (as a surrogate for sudden cardiac death). This study was conducted to assess the relationship between scar burden (extent and severity) and the follow-up left ventricular ejection fraction (LVEF). Methods. Patients were referred for viability assessment with late gadolinium enhancement (LGE) on cardiovascular magnetic resonance imaging.
The amount of replacement fibrosis in the LV myocardium (scar burden) measured by cardiac magnetic resonance (CMR) using late gadolinium enhancement imaging might be a key factor to individualize risk stratification and refine patient selection for mitral valve interventions (12–14). Necrosis regions found in the heart (including left atrium (LA) pre-ablation fibrosis, LA post-ablation scar and left ventricle (LV) infarction), depending on the location and size, can have various implications on the cardiac conditions of the patients.
Disease-causing genetic defects in arrhythmogenic cardiomyopathy according to predominant ventricular involvement. Mutations in DSP, PLN and FLNC genes are the most common gene defects causing arrhythmogenic left ventricular cardiomyopathy. LV, left .
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lv scar heart|myocardial scar and lvef
